The aim of intra-aortic counterpulsation is to balance myocardial oxygen supply to meet metabolic demands of the myocardium.
Supply refers to
·coronary artery anatomy
·diastolic pressure
·diastolic time
Demand refers to
·heart rate
·afterload
·preload
·contractility
Left ventricular failure is
la series of physiological changes occurringas a result of injury, which will result in an imbalance between supply and demand
lCO falls resulting in decreased supply
lpreload, afterload and heart rate increase in an attempt to compensate, resulting in increased demand
las LVF progresses, a greater imbalance between supply and demand results in further pump failure
Intra-aortic counter pulsation influences
·cardiac output
·systemic vascular resistance
·ejection fraction
IACP indications
lrefractory ventricular failure
lcardiogenic shock
lunstable refractory angina
limpending infarction
lmechanical complications due to acute myocardial infarction
lischaemia related intractable ventricular arrhythmias
lcardiac support for high risk general surgical patients and coronary angiography/angioplasty patients
lseptic shock
lweaning fromCPB
lsupport for failed angioplasty
IACP contra-indications
·severe aortic insufficiency
·abdominal aortic aneurysm
·severe calcified aorto-iliac disease
·peripheral vascular disease
·sheathless insertion not recommended in obese patients
IACPclinical considerations
Insertion
Catheter equipment
One sterile kit includes 1x18 gauge angiographic needle, guide wire, dilators x 2, introducer sheath with haemostasis valve, 60cc syringe, male luer lock plug, 90 cm pressure tubing, 1x datascope connector and the balloon catheter.
NB. Size is important. 40 cc catheter for patients > 5’4”(165 cm) and 35 cc catheter for patients <> 165 cm)
Monitoring
lMonitor half hourly HR MAP CVP PAP PCWP
lMonitor hourly
degree of diastolic augmentation
degree of afterload reduction
haemodynamic profile using cardiac output studies
neurovascular status
neurological status
renal function
insertion site for bleeding
lMonitor daily
biochemistry/ bleeding/ clotting studies
IABP
·maintain optimal augmentation and afterload reduction
·note placement of IABP on daily chest x-ray
·zero transducer 8 hourly and prn
·IABP autofills 2hrly
IABP management in cardiac arrest
*Patient can be safely defibrillated in the event of VT, VF with the IABP on standby
·select standby mode
·defibrillate according to protocol
·select assist mode
In the event of circulatory collapse necessitating external cardiac massage
·select pressure triggered mode
·the inflation trigger is the pressure generated be ECM
Special treatment needs
·note and record quality of pedal pulses 1 hourly
·utilize pressure reducing mattress
·maintain anti - coagulant protocol with low dose
heparinization
·observe for complications of IABC
Fluid balance management
l1 hourly assessment
Activity
·bed rest/log rolls
·do not elevate HOB > 30%
·do not flex balloon leg
·ROM uninvolved extremity
·dorsiflexion balloon leg
Diet
las tolerated
lsupplemental nutritional support
lN/E tube feeds
Triggering
The trigger for inflation is either the ECG, A-V or V pacing spike or the aortic pressure curve recorded at the tip of the IABC
ECG triggering
·preferably select the lead with the largest QRS complex
·persistent arrhythmias, tachyarrhythmias can make timing difficult
·efforts should be made to suppress arrhythmias and treat tachycardia
·in rapid atrial fibrillation position deflation point to extreme right
AV and V triggering:
·patient must be fully paced in this mode
·in intermittent pacing, select ECG trigger
Pressure triggering:
·use when atrial pacemaker spike interpreted as QRS complex
·use in transport when artifact interferes with triggering
Timing
Initially timing can be based on the ECG. The balloon is inflated at the peak of the T-wave (end systole) and deflated during the pr interval (end diastole). Fine tuning is then based on the aortic pressure waveform.
Inflation
·occurs at the dicrotic notch, indicative of aortic valve closure
Deflation
·occurs just before the aortic valve opens. Deflation should be timed to achieve the lowest presystolic pressure possible
Transport
Transport patients in the pressure wave triggering mode
Respiratory therapy
levaluate breath sounds
lroutine respiratory care
lmodified respiratory therapy
Daily/prn lab work
lmonitor K, se Cr, platelets, INR, clotting times
lCKMB, serial ECG
lABG 4hrly
lblood/urine/sputum cultures if T > 39
Weaning
When haemodynamics improve, inotropic support is < style="">
·weaning by decreasing the ratio of unassisted to augmented beats from 1:1 to 1:3
·weaning by decreasing the degree of balloon augmentation from100% to 30%
·anticoagulation to cease 4 hours prior to removal
·return to normal coagulation parameters
·patient to be independent of counterpulsation (30%augmentation) for at leastfive hours prior to removal of IABC
·serial haemodynamic profile to ascertain response to weaning
·observe clinical status to monitortoleranceto weaning
Removal
Removal of IABC necessitates return to theatre if balloon was inserted surgically under direct vision. Catheters inserted percutaneously, using the Seldinger technique, may be removed in the ICU setting using strict asepsis.
lIABP to standby
lballoon is deflated
lsheath and catheter removed simultaneously
lfirm pressure applied to insertion site for 30 minutes or until all bleeding ceases
lpressure bag applied to insertion site for further 4-6 hrs
lobservation of neurovascular status for 24 hours
Complications
Limb ischaemia:
lcheck distal pulses, colour, temperature 1 hourly
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